Security analysis of the micro transport protocol with a misbehaving receiver

BitTorrent is the most widely used Peer-to-Peer (P2P) protocol and it comprises the largest share of traffic in Europe. To make BitTorrent more Internet Service Provider (ISP) friendly, BitTorrent Inc. invented the Micro Transport Protocol (uTP). It is based on UDP with a novel congestion control called Low Extra Delay Background Transport (LEDBAT). This protocol assumes that the receiver always gives correct feedback, since otherwise this deteriorates throughput or yields to corrupted data. We show through experimental investigation that a misbehaving uTP receiver, which is not interested in data integrity, can increase the bandwidth of the sender by up to five times. This can cause a congestion collapse and steal large share of a victim’s bandwidth. We present three attacks, which increase the bandwidth usage significantly. We have tested these attacks in a real world environment and show its severity both in terms of number of packets and total traffic generated. We also present a countermeasure for protecting against the attacks and evaluate the performance of that defence strategy

Stealing bandwidth from BitTorrent seeders

BitTorrent continues to comprise the largest fraction of Internet traffic. While significant progress has been made in understanding the BitTorrent choking mechanism, its security vulnerabilities have not been investigated thoroughly. This paper presents an experimental analysis of bandwidth attacks against different choking algorithms in the BitTorrent seed state. We reveal a simple exploit that allows malicious peers to receive a considerably higher download rate than contributing leechers, therefore introducing significant efficiency degradations for benign peers. We show the damage caused by the proposed attack in two different environments: a lab testbed comprising 32 peers and a PlanetLab testbed with 300 peers. Our results show that 3 malicious peers can degrade the download rate up to 414.99% for all peers. Combined with a Sybil attack that consists of as many attackers as leechers, it is possible to degrade the download rate by more than 1000%. We propose a novel choking algorithm which is immune against bandwidth attacks and a countermeasure against the revealed attack

Voltammetry of (E)-l-Phenyl-2-Nitro-l-Propene in N,N-Dimethyiformamide Solutions

ABSTRACT The electrode reduction mechanism of (E)=phenyl-2-nitro-l-propene (PNP) in N,N-dimethylformamide (DMF) has been studied by polarographic, cyclic voltammetric, and rotating disk techniques. The compound is reduced in two polarographic steps in DMF and DMF-water solutions. Data taken at potentials of the first polarographic plateau suggest that the compound is reduced to a product via a mechanism involving coupling of the ion radicals of the precursor. The rate of the coupling reaction has been studied as a function of concentration of the PNP and water content of the solvent. Oscilloscopic recording of rapid cyclic voltammetry experiments demonstrate that the second polarographic step represents a two-electron reduction of the parent compound, PNP

RETRATO DE MUJER [Material gráfico]

Copia digital. Madrid : Ministerio de Educación, Cultura y Deporte, 201

Iron Behaving Badly: Inappropriate Iron Chelation as a Major Contributor to the Aetiology of Vascular and Other Progressive Inflammatory and Degenerative Diseases

The production of peroxide and superoxide is an inevitable consequence of aerobic metabolism, and while these particular "reactive oxygen species" (ROSs) can exhibit a number of biological effects, they are not of themselves excessively reactive and thus they are not especially damaging at physiological concentrations. However, their reactions with poorly liganded iron species can lead to the catalytic production of the very reactive and dangerous hydroxyl radical, which is exceptionally damaging, and a major cause of chronic inflammation. We review the considerable and wide-ranging evidence for the involvement of this combination of (su)peroxide and poorly liganded iron in a large number of physiological and indeed pathological processes and inflammatory disorders, especially those involving the progressive degradation of cellular and organismal performance. These diseases share a great many similarities and thus might be considered to have a common cause (i.e. iron-catalysed free radical and especially hydroxyl radical generation). The studies reviewed include those focused on a series of cardiovascular, metabolic and neurological diseases, where iron can be found at the sites of plaques and lesions, as well as studies showing the significance of iron to aging and longevity. The effective chelation of iron by natural or synthetic ligands is thus of major physiological (and potentially therapeutic) importance. As systems properties, we need to recognise that physiological observables have multiple molecular causes, and studying them in isolation leads to inconsistent patterns of apparent causality when it is the simultaneous combination of multiple factors that is responsible. This explains, for instance, the decidedly mixed effects of antioxidants that have been observed, etc...Comment: 159 pages, including 9 Figs and 2184 reference